Facebook
Research
References
- 2019
Ginsenoside compound K induces ROS-mediated apoptosis and autophagic inhibition in human neuroblastoma cells in vitro and in vivo. (Brain Cancer) - 2019Ginsenoside metabolite compound K induces apoptosis and autophagy in non-small cell lung cancer cells via AMPK-mTOR and JNK pathways. (Lung Cancer)
- 2018The ginsenoside metabolite compound K inhibits hormone-independent breast cancer through downregulation of cyclin D1. (Breast Cancer)
- 2018Ginsenoside compound K inhibits nuclear factor-кB by targeting Annexin A2. (Liver Cancer)
- 2017The ginsenoside metabolite compound K inhibits growth, migration and stemness of glioblastoma cells. (Brain Cancer)
- 2017Ginsenoside compound K enhances trailmediated renal cancer cell apoptosis through CHOP signaling. (Renal Cancer)
- 2017Effects of ginsenoside CK combined with 5-fluorouracil on the proliferation, apoptosis and epithelial mesenchymal transition of human pancreatic cancer PANC-1 cells. (Pancreatic Cancer)
- 2017Effects of compound K, an enteric microbiome metabolite of ginseng, in the treatment of inflammation associated colon cancer. (Colorectal Cancer)
- 2013Synthesis of esters of ginsenoside metabolite M1 and their cytotoxicity on MGC80-3 cells. (Gastric Cancer)
- 2013Anti-cancer effects of ginsenoside compound K on pediatric acute myeloid leukemia cells. (Leukemia)
- 2019
20(S)-Ginsenoside Rh2 displays efficacy against T-cell acute lymphoblastic leukemia through the PI3K/Akt/mTOR signal pathway. (Leukemia) - 2019Ginsenoside Rh2 Inhibits Angiogenesis in Prostate Cancer by Targeting CNNM1. (Prostate Cancer)
- 2019A stereo-selective growth inhibition profile of ginsenoside Rh2 on human colon cancer cells. (Colorectal Cancer)
- 2018Ginsenoside Rh2 inhibits human A172 glioma cell proliferation and induces cell cycle arrest status via modulating Akt signaling pathway. (Brain Cancer)
- 2017Ginsenoside Rh2 inhibited proliferation by inducing ROS mediated ER stress dependent apoptosis in lung cancer cells. (Lung Cancer)
- 2017Ginsenoside Rh2 epigenetically regulates cell-mediated immune pathway to inhibit proliferation of MCF-7 breast cancer cells. (Breast Cancer)
- 201720(S)-Ginsenoside Rh2 suppresses proliferation and migration of hepatocellular carcinoma cells by targeting EZH2 to regulate CDKN2A-2B gene cluster transcription. (Liver Cancer)
- 2017Ginsenoside Rh2 induces apoptosis and inhibits epithelial-mesenchymal transition in HEC1A and Ishikawa endometrial cancer cells. (Cancer of Corpus Uteri)
- 2017Ginsenoside Rh2 enhances the antitumor immunological response of a melanoma mice model. (Cutaneous Carcinoma)
- 2016Ginsenoside Rh2 inhibits proliferation and induces apoptosis of human gastric cancer SGC-7901 side population cells. (Gastric Cancer)
- 2019
Ginsenoside Rg3 inhibits growth and epithelial-mesenchymal transition of human oral squamous carcinoma cells by down-regulating miR-221. (Oral Cancer) - 2019Ginsenoside Rg3 inhibits cell growth, migration and invasion in Caco-2 cells by downregulation of lncRNA CCAT1. (Colorectal Cancer)
- 2019Effects of ginsenoside Rg3 on epigenetic modification in ovarian cancer cells. (Ovarian Cancer)
- 2019Regioselective synthesis and structures of anti-cancer 20(R)-ginsenoside Rg3 derivatives. (Pancreatic Cancer)
- 2019Ginsenoside Rg3 inhibits migration and invasion of nasopharyngeal carcinoma cells and suppresses epithelial mesenchymal transition. (Nasopharyngeal Carcinoma)
- 2019Ginsenoside Rg3 combined with oxaliplatin inhibits the proliferation and promotes apoptosis of hepatocellular carcinoma cells via downregulating PCNA and cyclin D1. (Liver Cancer)
- 2019Stereoselective anti-cancer activities of ginsenoside Rg3 on triple negative breast cancer cell models. (Breast Cancer)
- 2019Ginsenoside Rg3 suppresses the proliferation of prostate cancer cell line PC3 through ROS-induced cell cycle arrest. (Prostate Cancer)
- 2019Antitumor activity of ginsenoside Rg3 in melanoma through downregulation of the ERK and Akt pathways. (Cutaneous Carcinoma)
- 2017Ginsenoside Rg3 prevents oxidative stress-induced astrocytic senescence and ameliorates senescence paracrine effects on glioblastoma. (Brain Cancer)
